![JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4 JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4](https://www.spandidos-publications.com/article_images/etm/8/1/ETM-08-01-0153-g04.jpg)
JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4
![SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs13046-019-1467-6/MediaObjects/13046_2019_1467_Fig5_HTML.png)
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
![The JNK inhibitor SP600125 enhances dihydroartemisinin-induced apoptosis by accelerating Bax translocation into mitochondria in human lung adenocarcinoma cells - ScienceDirect The JNK inhibitor SP600125 enhances dihydroartemisinin-induced apoptosis by accelerating Bax translocation into mitochondria in human lung adenocarcinoma cells - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S0014579310006708-gr2.jpg)
The JNK inhibitor SP600125 enhances dihydroartemisinin-induced apoptosis by accelerating Bax translocation into mitochondria in human lung adenocarcinoma cells - ScienceDirect
![SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs13046-019-1467-6/MediaObjects/13046_2019_1467_Fig6_HTML.png)
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
![JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine](https://media.springernature.com/full/springer-static/image/art%3A10.3858%2Femm.2009.41.9.073/MediaObjects/12276_2009_Article_BFemm200975_Fig1_HTML.jpg)
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
![Effect of specific JNK inhibitor SP600125 (SP) on the effect of 1 mM... | Download Scientific Diagram Effect of specific JNK inhibitor SP600125 (SP) on the effect of 1 mM... | Download Scientific Diagram](https://www.researchgate.net/publication/235881092/figure/fig7/AS:316690488414220@1452516305089/Effect-of-specific-JNK-inhibitor-SP600125-SP-on-the-effect-of-1-mM-stearic-acid-SA-on.png)
Effect of specific JNK inhibitor SP600125 (SP) on the effect of 1 mM... | Download Scientific Diagram
![Effect of JNK inhibitor SP600125 on γH2AX production and cell viability... | Download Scientific Diagram Effect of JNK inhibitor SP600125 on γH2AX production and cell viability... | Download Scientific Diagram](https://www.researchgate.net/publication/5267201/figure/fig1/AS:340693546487817@1458239079981/Effect-of-JNK-inhibitor-SP600125-on-gH2AX-production-and-cell-viability-in-TrkA-induced.png)
Effect of JNK inhibitor SP600125 on γH2AX production and cell viability... | Download Scientific Diagram
![JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine](https://media.springernature.com/lw685/springer-static/image/art%3A10.3858%2Femm.2009.41.9.073/MediaObjects/12276_2009_Article_BFemm200975_Fig3_HTML.jpg)
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
![JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4 JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4](https://www.spandidos-publications.com/article_images/etm/8/1/ETM-08-01-0153-g03.jpg)
JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4
![IJMS | Free Full-Text | SP600125 Induces Src and Type I IGF Receptor Phosphorylation Independent of JNK IJMS | Free Full-Text | SP600125 Induces Src and Type I IGF Receptor Phosphorylation Independent of JNK](https://www.mdpi.com/ijms/ijms-15-16246/article_deploy/html/images/ijms-15-16246-g003.png)
IJMS | Free Full-Text | SP600125 Induces Src and Type I IGF Receptor Phosphorylation Independent of JNK
![JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine](https://media.springernature.com/lw685/springer-static/image/art%3A10.3858%2Femm.2009.41.9.073/MediaObjects/12276_2009_Article_BFemm200975_Fig4_HTML.jpg)
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
![JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine](https://media.springernature.com/lw685/springer-static/image/art%3A10.3858%2Femm.2009.41.9.073/MediaObjects/12276_2009_Article_BFemm200975_Fig6_HTML.jpg)
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
![SP600125, an inhibitor of c-jun N-terminal kinase, activates CREB by a p38 MAPK-mediated pathway - ScienceDirect SP600125, an inhibitor of c-jun N-terminal kinase, activates CREB by a p38 MAPK-mediated pathway - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S0006291X03012877-gr3.jpg)
SP600125, an inhibitor of c-jun N-terminal kinase, activates CREB by a p38 MAPK-mediated pathway - ScienceDirect
![JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4 JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4](https://www.spandidos-publications.com/article_images/etm/8/1/ETM-08-01-0153-g00.jpg)
JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4
![JNK inhibitor SP600125 enhances TGF-β-induced apoptosis of RBE human cholangiocarcinoma cells in a Smad-dependent manner. | Semantic Scholar JNK inhibitor SP600125 enhances TGF-β-induced apoptosis of RBE human cholangiocarcinoma cells in a Smad-dependent manner. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/2d73e2ba286d28443dcf275815386208078be6f2/5-Figure4-1.png)